2009 H1N1 Virus Transmission and Outbreaks

T. M. Uyeki
Epidemiology and Prevention Branch, Influenza Division, National Center for Immunization and Respiratory Diseases,
Centers for Disease Control and Prevention, Atlanta


The New England Journal of Medicine
NEJM Volume 362:2175-2184  June 10, 2010  Number 23

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The emergence of 2009 pandemic influenza A (H1N1) virus prompted early questions about how the virus was spreading and how easily it was transmitted, along with uncertainty about disease severity, clinical complications and risk factors for severe disease, and the effectiveness of antiviral treatment and control measures. Without a vaccine against the 2009 H1N1 virus, early control measures included both pharmaceutical interventions (the use of antiviral agents) and nonpharmaceutical interventions (e.g., school closures, isolation, and quarantine), depending on the specific outbreak setting, available resources, and goals. The use of these interventions varied according to country and the evolution of the pandemic. It was determined quickly that the 2009 H1N1 virus was resistant to the adamantane antiviral agents but susceptible to neuraminidase inhibitors (oseltamivir and zanamivir),1 which could be used for treatment and chemoprophylaxis.2

In this issue of the Journal, findings of two studies that were conducted during the early phase of the pandemic are reported, with implications for the control of influenza outbreaks



Oseltamivir Ring Prophylaxis for Containment of 2009 H1N1 Influenza Outbreaks

V. J. Lee and Others
Abstract | FREE Full Text | PDF  | Supplementary Material | 
Conclusions Oseltamivir ring chemoprophylaxis, together with prompt identification and isolation of infected personnel, was effective in reducing the impact of outbreaks of 2009 H1N1 influenza in semiclosed settings

Comparative Epidemiology of Pandemic and Seasonal Influenza A in Households

B. J. Cowling and Others
Abstract | FREE Full Text | PDF  | Supplementary Material | 
Pandemic 2009 H1N1 virus has characteristics that are broadly similar to those of seasonal influenza A viruses in terms of rates of viral shedding, clinical illness, and transmissibility in the household setting.  

 





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